Angina, Coronary Insufficiency, And Myocardial Infarction

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Angina, Coronary Insufficiency, And Myocardial infarction

All of the above are sometimes used synonymously with the phrase "heart attack", they are quite different. Patients with coronary artery disease ease experience angina when the flow of blood is not sufficient to meet the needs of the heart at that moment. This occurs when the heart has to work harder because of physical exertion, or spasm in a coronary artery reduces oxygen-rich blood flow. Anginal chest pain is transient, and usually relieved by nitroglycerin or other drugs which dilate coronary vessels. It is more apt to occur when??? patients are anemic and have fewer red cells m carry oxygen, or at high altitudes, where red cells can't pick up as much oxygen as they pass through the lungs because of lower atmospheric concentrations. When the imbalance between the heart's requirement for oxygen and its availability persists, chest pain is apt be more severe and prolonged, a condition known as coronary insufficiency.

If this problem is not resolved rapidly enough, a myocardial infarction will occur, meaning that some of the affected heart muscle tissue will die. The degree and extant of damage is reflected in the electrocardiogram, and can be visualized with various sophisticated imaging procedures. During episodes of angina and coronary insufficiency, the electrocardiogram and other tests can also demonstrate deficiencies in the delivery of oxygen to heart muscle. However, thee abnormal findings are only temporary, and vanish following the attack. Patients can have multiple episodes of angina for years, and still have normal electrocardiograms when they are symptom free, because any damage is completely reversible.

During a myocardial infarction, some heart cells die, and adjacent areas may subsequently suffer various degrees of permanent or sustained damage, depending on the degree of deprived blood flow. However, even healthy heart muscle with normal circulation is affected, since the inflammation, swelling, and waste products resulting from dead and dying tissue can interfere with their nourishment. Microscopic analysis reveals a central core of dead cells, surrounded by a zone of severe damage that could become permanent, which blends into a still larger area of more peripheral tissue that is progressively less affected, but still not functioning optimally.

Much of the injury from an acute myocardial infarction can be prevented or reversed by rapidly supplying adequate nutrition and oxygen to cells that have not yet died. As will be expiated, polarizing solution is able m accomplish this for several reasons.

The American Institute of Stress.

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